Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis

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Science Immunology  14 Apr 2017:
Vol. 2, Issue 10, eaag3358
DOI: 10.1126/sciimmunol.aag3358

Article Information

vol. 2 no. 10

Published By: 
  • Received June 10, 2016
  • Revision received January 15, 2017
  • Accepted March 6, 2017

Author Information

  1. Carmelo Carmona-Rivera1,
  2. Philip M. Carlucci1,
  3. Erica Moore1,
  4. Nithya Lingampalli2,3,
  5. Hannes Uchtenhagen4,
  6. Eddie James4,
  7. Yudong Liu1,
  8. Kevin L. Bicker5,
  9. Heidi Wahamaa6,
  10. Victoria Hoffmann7,
  11. Anca Irinel Catrina6,
  12. Paul R. Thompson8,
  13. Jane H. Buckner4,
  14. William H. Robinson2,3,
  15. David A. Fox9 and
  16. Mariana J. Kaplan1,*
  1. 1Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD 20892, USA.
  2. 2VA Palo Alto Health Care System, Palo Alto, CA 94304, USA.
  3. 3Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA 94305, USA.
  4. 4Translational Research Program, Benaroya Research Institute at Virginia Mason, Seattle, WA 98101, USA.
  5. 5Department of Chemistry, Middle Tennessee State University, 1301 East Main Street, Murfreesboro, TN 37132, USA.
  6. 6Department of Medicine, Solna, Karolinska University Hospital, Stockholm S17176, Sweden.
  7. 7Division of Veterinary Resources, Office of the Director, National Institutes of Health, Bethesda, MD 20892, USA.
  8. 8Department of Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, Worcester, MA 01605, USA.
  9. 9Division of Rheumatology, Department of Internal Medicine, University of Michigan, Ann Arbor, MI 48109, USA.
  1. *Corresponding author. Email: mariana.kaplan{at}


Article usage

Apr 2017277088158
May 201711274065

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