Science Immunology

Supplementary Materials

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  • Material and Methods
  • Fig. S1. scRNA-seq analysis of T cells from human blood and healthy kidney tissue from tumor nephrectomies.
  • Fig. S2. Profiling of T cells from human blood and healthy kidney tissue from tumor nephrectomies.
  • Fig. S3. scRNA-seq analysis of T cells from blood and kidney of patients with ANCA-GN.
  • Fig. S4. Profiling of T cells from kidney and blood of patients with ANCA-GN.
  • Fig. S5. Flow cytometry gating strategy for murine T cell analysis.
  • Fig. S6. Prior L. monocytogenes infection does not affect crescentic GN.
  • Fig. S7. Effect of local S. aureus skin infection on renal TRM17 cell generation and effect of systemic S. aureus infection on imiquimod-induced skin inflammation.
  • Fig. S8. Gene expression analysis of renal CD4+ T cells from nephritic mice previously infected with S. aureus.
  • Fig. S9. The TH17-associated pathogens C. albicans and E. coli cause renal TRM17 cell formation.
  • Fig. S10. Conceptual link of pathogen-induced tissue resident memory T cells with autoimmunity.
  • Table S1. Baseline characteristics of controls at time of biopsy.
  • Table S2. Baseline characteristics of patients with ANCA-GN at the time of biopsy.
  • Table S3. Clinical characteristics and ANCA-serotype of patients from the Hamburg GN Registry.
  • Table S4. List of transcription factors identified by TRRUSTv2 analysis.
  • References (6879)

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